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anti phosphosrc tyr416  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc anti phosphosrc tyr416
    Anti Phosphosrc Tyr416, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/phosphosrc+tyr416/10__21203_slash_rs__3__rs___6855159_slash_v1-172-14-17
    Average 86 stars, based on 1 article reviews
    anti phosphosrc tyr416 - by Bioz Stars, 2026-10
    86/100 stars

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    Related Articles

    other:

    Article Title: Stimulation of extracellular signal-regulated kinases and proliferation in the human gastric cancer cells KATO-III by obestatin.
    Article Snippet: Obestatin, the ghrelin-associated peptide, activates cell proliferation in the gastric cancer cell line KATO-III.. The results showed that this peptide induced cell proliferation by mitogen-activated kinase kinase/extracellular signal-regulated kinases1/2 (ERK1/2) phosphorylation.. A sequential analysis of the obestatin transmembrane signalling pathway indicated that the ERK1/2 activity is partially blocked after preincubation of the cells with pertussis toxin, as well as by wortmannin (an inhibitor of phosphoinositide 3-kinase (PI3K)), staurosporine (an inhibitor of protein kinase C (PKC)) and 4-amino-5(4-chlorophenyl)-7-(t-butyl)pyrazolo[3,4-d]pyrimidine (PP2, which inhibits the non receptor tyrosine kinase Src).

    Article Title: Silencing Delta-like 1 Expression Induces Migratory Features in Pancreatic Cancer Cells Through Stimulation of Src and p38 Signalling Pathway.
    Article Snippet: Background/Aim: The prognosis of pancreatic cancer has not improved due to its migratory feature and refractory potential to chemo-resistance with absence of effective diagnosis.. Despite continuous efforts, its underlying mechanisms of malignant nature remain ambiguous.. The objective of this study was to investigate delta-like 1 (DLL1) as a tumor suppressor in the metastasic ability of human pancreatic cancer cells.

    Transduction:

    Article Title: LH and FSH promote migration and invasion properties of a breast cancer cell line through regulatory actions on the actin cytoskeleton.
    Article Snippet: Accepted Manuscript LH and FSH promote migration and invasion properties of a breast cancer cell line through regulatory actions on the actin cytoskeleton Angel Matias Sanchez, Marina Ines Flamini, Eleonora Russo, Elena Casarosa, Simone Pacini, Mario Petrini, Andrea Riccardo Genazzani, Tommaso Simoncini PII: S0303-7207(16)30299-4 DOI: 10.1016/j.mce.2016.08.009 Reference: MCE 9603 To appear in: Molecular and Cellular Endocrinology Received Date: 2 December 2015 Revised Date: 19 July 2016 Accepted Date: 5 August 2016 Please cite this article as: Sanchez, A.M., Flamini, M.I., Russo, E., Casarosa, E., Pacini, S., Petrini, M., Genazzani, A.R., Simoncini, T., LH and FSH promote migration and invasion properties of a breast cancer cell line through regulatory actions on the actin cytoskeleton, Molecular and Cellular Endocrinology (2016), doi: 10.1016/j.mce.2016.08.009.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript.

    Incubation:

    Article Title: Involvement of glucagon-like peptide-1 receptor in fisetin tetramethyl ether-enhanced insulin secretion in MIN6 cells.
    Article Snippet: Background: Pongamia pinnata (L.) Pierre, long used as a traditional medicine to treat diabetes and metabolic disorders, shows an insulinotropic effect.. Objective: This study aimed to determine the insulinotropic property and underlying mechanism of a constituent of Pongamia pinnata, fisetin tetramethyl ether (FTM).. Methods: The insulinotropic property of FTM was investigated using pancreatic beta cell line MIN6, while secreted insulin and residual insulin were detected by an insulin assay.



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